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Table 1 Top 5 pathways enriched by FunRich 3.0. In comparing controls (CTRL) vs Low-garde (LG) and High-grade dysplasia (HG) and pancreatic carcinoma (CAR), the following pathways were found to be enriched. The pathways are given in the order of their degree of enrichment from top to bottom in every column. HM means highest mean

From: Label-free serum proteomics and multivariate data analysis identifies biomarkers and expression trends that differentiate Intraductal papillary mucinous neoplasia from pancreatic adenocarcinoma and healthy controls

1. CTRL VS LG 2. CTRL VS HG 3. CTRL VS CAR
1A. HMCTRL 1B. HMLG 2A. HMCTRL 2B. HMHG 3A. HMCTRL 3B. HMCAR
1A1.Hemostasis 1B1.Integrin family cell surface interactions 2A1.Formation of Fibrin Clot (Clotting Cascade) 2B1.Complement cascade 3A1.Hemostasis 3B1.Complement cascade
1A2.Platelet activation, signaling and aggregation 1B2.Beta1 integrin cell surface interactions 2A2.Hemostasis 2B2.Initial triggering of complement 3A2.Complement cascade 3B2.Formation of Fibrin Clot (Clotting Cascade)
1A3.Initial triggering of complement 1B3.Proteoglycan syndecan-mediated signaling events 2A3.Initial triggering of complement 2B3.Innate Immune System 3A3.Initial triggering of complement 3B3.Initial triggering of complement
1A4.Intrinsic Pathway 1B4.Alpha9 beta1 integrin signaling events 2A4.Complement cascade 2B4.Formation of Fibrin Clot (Clotting Cascade) 3A4.Formation of Fibrin Clot (Clotting Cascade) 3B4.Intrinsic Pathway
1A5.Complement cascade 1B5.Glypican pathway 2A5.Intrinsic Pathway 2B5.Terminal pathway of complement 3A5.Intrinsic Pathway 3B5.Innate Immune System
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Translational Medicine Communications

ISSN: 2396-832X

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